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Cerebral Apoplexy

Chirwere

Cerebral Apoplexy

Cerebral Stroke, inozivikanwawo seCerebral Apoplexy, yaona kuwedzera kwezviitiko nekuda kwekuchinja kwechikafu chemazuva ano uye mararamiro. Yakava imwe yematambudziko makuru anokanganisa hutano hwevanhu. Nekufambira mberi mune zvekurapa, varwere vazhinji vesitiroko vanogona kuwedzera hupenyu hwavo kuburikidza nekurapa kwakakodzera uye nenguva. Nekudaro, varwere ava vanowanzoona zvakasara sequelae sekuremara kwemakumbo, kukuvara kwemota, kukanganiswa kwemutauro, nezvimwewo, nekuda kwekukuvadzwa kweiyo central nerve system, zvakanyanya kukanganisa hupenyu hwevarwere uye kuvaremedza zvakanyanya mumuviri nepfungwa. mhuri, uye nzanga.

    Etiology

    Zvikonzero chaizvo zvesitiroko hazvisati zvanyatsojeka. Parizvino njodzi dzakaonekwa dzinokonzera sitiroko sequelae dzinosanganisira: hypertension, kufutisa, kuputa uye kunwa doro, chirwere chemoyo, mhando yemvura, genetics, kudya munyu, pakati pezvimwe, izvo zvinotungamira kune zvinotevera zvinotevera.

    Cerebral Apoplexya2p

    Hypertension

    Hypertension ndiyo inonyanya kuisa njodzi kune zvese ischemic uye hemorrhagic sitiroko, sezvo paine hukama hwemutsara pakati pemazinga eBP uye njodzi yesitiroko, hukama hwakasimbiswa nemakore ekutsvagisa.

    Chirwere cheMwoyo

    Kusashanda zvakanaka kwemoyo hakungokonzeresa kukonzeresa hypertension yenguva refu uye kukuvadza vascular system asi kunogona kukonzera zvakananga sitiroko.

    Chirwere cheshuga

    Iko kuwirirana pakati pekiriniki yechirwere cheshuga nesitiroko ndeyechokwadi. Kunyangwe zvinyoro zveglucose metabolism kusagadzikana kunoratidza kuwedzera kwengozi yeischemic sitiroko. Ngozi yesitiroko muvarwere vane chirwere cheshuga yakakura muvakadzi pane varume.

    Kufutisa

    Kufutisa ndiyo ngozi huru yehypertension uye chirwere cheshuga. Inobatanidzwa nekuchinja kwehuremu hwemuviri uye kuwedzera kwecholesterol uye triglycerides muropa, zvichiratidza mukana wengozi yekurohwa.

    Kusvuta

    Kusvuta kwakabatana nesitiroko. Ngozi yekuitika sitiroko inopeta katatu muvanoputa zvakanyanya zvichienzaniswa nevasingasvuti.

    Clinical Manifestations

    Makiriniki ekuratidzwa kwesitiroko sequelae anosiyana pakati pevarwere nenzvimbo dzakakanganisika, kusanganisira:

    Kuremara kwemotokari: Kuoma mitezo, kushaya simba kwetsandanyama, uye kusashanda zvakanaka kwemotokari.

    Kurasikirwa kwekunzwa: Kuderedzwa kana kurasikirwa tactile, maonero ekushisa, uye proprioception.

    Kukanganiswa kwekutaura: Aphasia, kuderedzwa kwekutaura, uye matambudziko ekunzwisisa mutauro.

    Kukanganisika kwepfungwa: Kuderera mundangariro, kushaikwa kwekuisa pfungwa pachinhu chimwe, uye kufunga kunononoka.

    Kukanganiswa kwekuona: Kurasikirwa kwekuona uye kukanganiswa kwenzvimbo yekuona.

    Examination

    Neuroimaging zvidzidzo: Brain MRI, CT scans kuti ione sitiroko yerudzi, nzvimbo, uye chiyero.

    Electroencephalogram (EEG): Inotarisa huropi hwemagetsi, inoona kubatwa, uye zvimwe zvisizvo.

    Kuongorora ropa: Kunzwisisa mamiriro eropa remurwere, kusasanganisa zvimwe zvinokonzeresa.

    Neurological bvunzo: Kuongorora nzvimbo dzakakanganisika kuburikidza nekuongororwa kwemota, sensory, uye reflex mabasa.

    Kuongororwa

    Kuongororwa kunowanzoenderana nenhoroondo yekurapa, kuratidzwa kwekiriniki, uye mhedzisiro yekuongororwa kwakakodzera. Vanachiremba vanosarudza kuongororwa zvichienderana nerudzi rwesitiroko, nzvimbo dzakakanganisika, uye maitiro eiyo sequelae.

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